viral and infectious

These help group studies by possible triggers: Epstein–Barr Virus Research examining EBV reactivation, antibody responses, and viral persistence in Long COVID and related conditions. Viral Reactivation Studies exploring reactivation of latent viruses following COVID infection. SARS-CoV-2 Persistence Research investigating whether viral remnants/reservoirs persist in the body after acute infection. Herpesviruses Studies involving EBV, HHV-6, CMV, or other herpesviruses in post-viral illness.
20
Feb
Potential Bartonella Reactivation Associated With Long COVID

Potential Bartonella Reactivation Associated With Long COVID

A case study reported that Bartonella henselae infection appeared to reactivate following acute COVID-19, suggesting that immune changes associated with SARS-CoV-2 infection may influence the clinical behavior of latent bacterial infections.
2 min read
21
Jan
Intestinal barrier compromise, viral persistence, and immune dysregulation converge on neurological sequelae in Long COVID

Intestinal barrier compromise, viral persistence, and immune dysregulation converge on neurological sequelae in Long COVID

A new review in Frontiers in Aging Neuroscience outlines how Long COVID may be driven by a combination of persistent viral material, immune system dysregulation, gut barrier disruption, abnormal blood clotting, and inflammation in the brain.
2 min read
15
Jan
Increased phosphorylated tau (pTau-181) is associated with neurological post-acute sequelae of coronavirus disease in essential workers: a prospective cohort study before and after COVID-19 onset

Increased phosphorylated tau (pTau-181) is associated with neurological post-acute sequelae of coronavirus disease in essential workers: a prospective cohort study before and after COVID-19 onset

New longitudinal data from essential workers suggests a neurological Long COVID subtype is associated with a marked rise in plasma pTau-181 (a biomarker often used in Alzheimer’s disease research) compared with pre-infection baseline.
3 min read
12
Jan
Virus-induced endothelial senescence as a cause and driving factor for ME/CFS and long COVID: mediated by a dysfunctional immune system

Virus-induced endothelial senescence as a cause and driving factor for ME/CFS and long COVID: mediated by a dysfunctional immune system

A new theory suggests some viral infections may push blood vessel cells into a long-term stress state, offering a possible explanation for the lasting symptoms seen in ME/CFS and Long COVID.
3 min read
12
Jan
Black-and-white electron micrograph showing two Epstein–Barr virus particles with round capsids surrounded by a membrane envelope.

Patients Suffering From Post-COVID-19 Syndrome Feature Enhanced Antibody Reactivity Towards Specific Linear Epitopes Within EBV EBNA1.

Research shows that Post COVID patients had stronger immune responses to certain parts of the Epstein-Barr virus protein, suggesting a possible link to Long COVID.
1 min read
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