COVID-19-induced gastrointestinal autonomic dysfunction: A systematic review
A sweeping new systematic review finds that COVID-19 may disrupt the autonomic nervous system—the body’s “automatic control” network that helps run digestion—potentially driving persistent gut symptoms in some people with Long COVID.
Increased phosphorylated tau (pTau-181) is associated with neurological post-acute sequelae of coronavirus disease in essential workers: a prospective cohort study before and after COVID-19 onset
New longitudinal data from essential workers suggests a neurological Long COVID subtype is associated with a marked rise in plasma pTau-181 (a biomarker often used in Alzheimer’s disease research) compared with pre-infection baseline.
Virus-induced endothelial senescence as a cause and driving factor for ME/CFS and long COVID: mediated by a dysfunctional immune system
A new theory suggests some viral infections may push blood vessel cells into a long-term stress state, offering a possible explanation for the lasting symptoms seen in ME/CFS and Long COVID.
Patients Suffering From Post-COVID-19 Syndrome Feature Enhanced Antibody Reactivity Towards Specific Linear Epitopes Within EBV EBNA1.
Research shows that Post COVID patients had stronger immune responses to certain parts of the Epstein-Barr virus protein, suggesting a possible link to Long COVID.
